NephBytes delivers concise, clinically focused audio summaries of essential nephrology textbooks — designed for fellows, residents, attendings, and anyone who wants to master renal medicine but doesn't have time to sit down with a 1,200-page book. Each episode breaks down high-yield concepts from core texts like Comprehensive Clinical Nephrology and Renal Physiology, with clinical vignettes, bedside pearls, and board-relevant takeaways — all in 20 minutes or less. This isn't a textbook read aloud. It's the version your favorite attending would teach you on a quiet call night.
Podcast Overview
NephBytes delivers concise, clinically focused audio summaries of essential nephrology textbooks — designed for fellows, residents, attendings, and anyone who wants to master renal medicine but doesn't have time to sit down with a 1,200-page book. Each episode breaks down high-yield concepts from core texts like Comprehensive Clinical Nephrology and Renal Physiology, with clinical vignettes, bedside pearls, and board-relevant takeaways — all in 20 minutes or less. This isn't a textbook read aloud. It's the version your favorite attending would teach you on a quiet call night.
Language
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Publishing Since
2/13/2026
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Recent Episodes
February 27, 2026
The Special Populations: Cirrhosis, Dialysis, and Everything You Need to Remember
<p>The final episode of the hyponatremia series. Covers hypervolemic hyponatremia in cirrhosis — including the potassium-first approach — asymptomatic cirrhotic hyponatremia, and severe hyponatremia requiring emergent dialysis and why CVVH with modified replacement fluid is the only safe option. Closes with a rapid-fire review of all 12 clinical pearls from the series.</p><p><br></p>
February 27, 2026
When ADH Is Off But Sodium Still Falls: Water Intoxication, Exercise Hyponatremia, and the Low Solute Trap
<p>Every episode so far has been about ADH being on when it shouldn't be. Today we flip that entirely.</p><p>This episode covers hyponatremia where ADH is suppressed, the urine is maximally dilute, and the kidneys are doing exactly what they're supposed to — but the patient is still hyponatremic. Either the water intake is overwhelming the kidneys' capacity, or there isn't enough solute to carry the water out.</p><p>We work through four presentations: acute water intoxication from a college drinking contest, where the critical teaching point is that rapid correction is safe in acute hyponatremia (unlike chronic); exercise-associated hyponatremia in a marathon runner, including field treatment with hypertonic saline and why normal saline makes things worse; tea-and-toast hyponatremia, with the solute math that explains why a patient eating almost nothing can develop hyponatremia even with maximally dilute urine and normal ADH; and primary polydipsia, where the diagnosis is pure volume overwhelming a compromised excretory system.</p><p>A three-question framework at the close ties the group together: is intake overwhelming capacity, is solute too low, or is excretory capacity compromised?</p><p>Final episode of the hyponatremia series next: cirrhosis and dialysis — the special populations where the standard algorithm breaks down.</p>
February 23, 2026
Salt Wasting: The Diagnosis That Looks Like SIADH But Requires the Opposite Treatment
<p>Same low osmolality. Same concentrated urine. Same high urine sodium. A clinician scanning those results would call it SIADH without hesitating — and then reach for fluid restriction. In salt wasting, that's exactly the wrong move.</p><p><br></p><p>This episode covers three salt wasting syndromes where the confusion with SIADH causes real harm. First, cerebral salt wasting after subarachnoid hemorrhage — how to distinguish it from SIADH using volume status, fractional excretion of phosphorus, and the saline challenge, which functions as both a diagnostic test and a therapeutic intervention. Second, cisplatin-induced renal salt wasting — a tubular toxicity that causes massive ongoing sodium losses (up to 400 mEq/day) in oncology patients, and why the combination of polyuria, hypovolemia, and a high urine sodium should never be attributed to SIADH. Third, vomiting-associated hyponatremia — why the urine sodium lies in the setting of metabolic alkalosis, why the urine chloride tells the truth, and how bicarbonaturia drags sodium into the urine even in a truly volume-depleted patient.</p><p><br></p><p>The three-question framework at the close gives you a systematic way to work through any case where the urine sodium is high and SIADH is on your differential.</p><p><br></p><p>Next episode: water intoxication, exercise-associated hyponatremia, and the low solute syndromes — where ADH is suppressed and the kidneys are doing everything right, but the patient is still hyponatremic.</p><p></p>
8 total episodes available
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Frequently asked questions
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- What is NephBytes?
- How often does this podcast release new episodes?
This podcast updates daily.
- Where can I listen to this podcast?
This podcast is available on 4 platforms including Apple Podcasts, Spotify, and more. You can also use the RSS feed directly.
- Does this podcast accept guests?
No, this podcast does not typically feature guests.
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